Selected Publications: Shraddha Sapkota

 

Sapkota, S., & Dixon, R.A. (2018). A network of genetic effects on non-demented cognitive aging: Alzheimer’s genetic risk (CLU + CR1 + PICALM) intensifies cognitive aging genetic risk (COMT + BDNF) selectively for APOE ε4 carriers. Journal of Alzheimer's Disease, 62(2), 887-900. doi: 10.3233/JAD-170909

 

Sapkota, S., Huan, T., Tran, T., Zheng, J., Camicioli, R., Li, L., & Dixon, R.A. (2018). Alzheimer's biomarkers from multiple modalities selectively discriminate clinical status: Relative importance of salivary metabolomics panels, genetic, lifestyle, cognitive, functional health, and demographic risk markers. Frontiers in Aging Neuroscience, 10, 296. doi: 10.3389/fnagi.2018.00296

 

Sapkota, S., Bäckman, L., & Dixon, R.A. (2017). Executive function performance and change in aging is predicted by apolipoprotein E, intensified by catechol-O-methyltransferase and brain-derived neurotrophic factor, and moderated by age and lifestyle. Neurobiology of Aging, 52, 81-89. doi: 10.1016/j.neurobiolaging.2016.12.022

 

Sapkota, S., Wiebe, S.A., Small, B.J., & Dixon, R.A. (2016). Apolipoprotein E and clusterin can magnify effects of personality vulnerability on declarative memory performance in non-demented older adults. International Journal of Geriatric Psychiatry, 31(5), 502-509. doi: 10.1002/gps.4355

 

Sapkota, S., Vergote, D., Westaway, D., Jhamandas, J., & Dixon, R.A. (2015). Synergistic associations of catechol-O-methyltransferase and brain-derived neurotrophic factor with executive function in aging are selective and modified by apolipoprotein E. Neurobiology of Aging, 36, 249-256. doi:10.1016/j.neurobiolaging.2014.06.020

 

Sapkota, S., Gee, M., Sabino, J., Emery, D., & Camicioli, R. (2014). Association of homocysteine on ventricular dilatation and brain atrophy in Parkinson’s disease. Movement Disorders, 29(3), 368-374. doi:10.1002/mds.25798